Imperial College London
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Mr(Dr) Chinmay Gupte

Faculty of Medicine, Department of Surgery & Cancer

Reader in Orthopaedics
 
 
 
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Contact

 

+44 (0)20 8846 1234 ext 15215c.gupte00 Website

 
 
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Location

 

Charing Cross Hospital 7 East wingCharing Cross HospitalCharing Cross Campus

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Summary

 

Publications

Citation

BibTex format

@article{Kittl:2016:10.1177/0363546515614312,
author = {Kittl, C and El-Daou, H and Athwal, KK and Gupte, CM and Weiler, A and Williams, A and Amis, AA},
doi = {10.1177/0363546515614312},
journal = {AMERICAN JOURNAL OF SPORTS MEDICINE},
pages = {345--354},
title = {The role of the anterolateral structures and the ACL in controlling laxity of the intact and ACL-deficient knee},
url = {http://dx.doi.org/10.1177/0363546515614312},
volume = {44},
year = {2016}
}
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RIS format (EndNote, RefMan)

TY  - JOUR
AB  - Background:Anterolateral rotatory instability (ALRI) may result from combined anterior cruciate ligament (ACL) and lateral extra-articular lesions, but the roles of the anterolateral structures remain controversial.Purpose:To determine the contribution of each anterolateral structure and the ACL in restraining simulated clinical laxity in both the intact and ACL-deficient knee.Study Design:Controlled laboratory study.Methods:A total of 16 knees were tested using a 6 degrees of freedom robot with a universal force-moment sensor. The system automatically defined the path of unloaded flexion/extension. At different flexion angles, anterior-posterior, internal-external, and internal rotational laxity in response to a simulated pivot shift were tested. Eight ACL-intact and 8 ACL-deficient knees were tested. The kinematics of the intact/deficient knee was replayed after transecting/resecting each structure of interest; therefore, the decrease in force/torque reflected the contribution of the transected/resected structure in restraining laxity. Data were analyzed using repeated-measures analyses of variance and paired t tests.Results:For anterior translation, the intact ACL was clearly the primary restraint. The iliotibial tract (ITT) resisted 31% ± 6% of the drawer force with the ACL cut at 30° of flexion; the anterolateral ligament (ALL) and anterolateral capsule resisted 4%. For internal rotation, the superficial layer of the ITT significantly restrained internal rotation at higher flexion angles: 56% ± 20% and 56% ± 16% at 90° for the ACL-intact and ACL-deficient groups, respectively. The deep layer of the ITT restrained internal rotation at lower flexion angles, with 26% ± 9% and 33% ± 12% at 30° for the ACL-intact and ACL-deficient groups, respectively. The other anterolateral structures provided no significant contribution. During the pivot-shift test, the ITT provided 72% ± 14% of the restraint at 45° for th
AU  - Kittl,C
AU  - El-Daou,H
AU  - Athwal,KK
AU  - Gupte,CM
AU  - Weiler,A
AU  - Williams,A
AU  - Amis,AA
DO  - 10.1177/0363546515614312
EP  - 354
PY  - 2016///
SN  - 0363-5465
SP  - 345
TI  - The role of the anterolateral structures and the ACL in controlling laxity of the intact and ACL-deficient knee
T2  - AMERICAN JOURNAL OF SPORTS MEDICINE
UR  - http://dx.doi.org/10.1177/0363546515614312
UR  - http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000369983300011&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=1ba7043ffcc86c417c072aa74d649202
UR  - https://journals.sagepub.com/doi/10.1177/0363546515614312
UR  - http://hdl.handle.net/10044/1/61286
VL  - 44
ER  -
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