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BibTex format

@article{Hayee:2011:glycob/cwr023,
author = {Hayee, B and Antonopoulos, A and Murphy, EJ and Rahman, FZ and Sewell, G and Smith, BN and McCartney, S and Furman, M and Hall, G and Bloom, SL and Haslam, SM and Morris, HR and Boztug, K and Klein, C and Winchester, B and Pick, E and Linch, DC and Gale, RE and Smith, AM and Dell, A and Segal, AW},
doi = {glycob/cwr023},
journal = {GLYCOBIOLOGY},
pages = {914--924},
title = {<i>G6PC3</i> mutations are associated with a major defect of glycosylation: a novel mechanism for neutrophil dysfunction},
url = {http://dx.doi.org/10.1093/glycob/cwr023},
volume = {21},
year = {2011}
}

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RIS format (EndNote, RefMan)

TY  - JOUR
AU  - Hayee,B
AU  - Antonopoulos,A
AU  - Murphy,EJ
AU  - Rahman,FZ
AU  - Sewell,G
AU  - Smith,BN
AU  - McCartney,S
AU  - Furman,M
AU  - Hall,G
AU  - Bloom,SL
AU  - Haslam,SM
AU  - Morris,HR
AU  - Boztug,K
AU  - Klein,C
AU  - Winchester,B
AU  - Pick,E
AU  - Linch,DC
AU  - Gale,RE
AU  - Smith,AM
AU  - Dell,A
AU  - Segal,AW
DO  - glycob/cwr023
EP  - 924
PY  - 2011///
SN  - 0959-6658
SP  - 914
TI  - <i>G6PC3</i> mutations are associated with a major defect of glycosylation: a novel mechanism for neutrophil dysfunction
T2  - GLYCOBIOLOGY
UR  - http://dx.doi.org/10.1093/glycob/cwr023
UR  - https://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000291526700006&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=a2bf6146997ec60c407a63945d4e92bb
VL  - 21
ER  -

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Emeritus Professor Howard R. Morris FRS

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