Publications
73 results found
Halliday BP, Chiew K, Newsome S, et al., 2017, Incremental prognostic value of cardiopulmonary exercise testing in non-ischemic dilated cardiomyopathy, EUROPEAN JOURNAL OF HEART FAILURE, Vol: 19, Pages: 435-435, ISSN: 1388-9842
Pennell DJ, Baksi AJ, Prasad SK, et al., 2016, Review of Journal of Cardiovascular Magnetic Resonance 2015, Journal of Cardiovascular Magnetic Resonance, Vol: 18, Pages: 86-86, ISSN: 1097-6647
There were 116 articles published in the Journal of Cardiovascular Magnetic Resonance (JCMR) in 2015, which is a 14 % increase on the 102 articles published in 2014. The quality of the submissions continues to increase. The 2015 JCMR Impact Factor (which is published in June 2016) rose to 5.75 from 4.72 for 2014 (as published in June 2015), which is the highest impact factor ever recorded for JCMR. The 2015 impact factor means that the JCMR papers that were published in 2013 and 2014 were cited on average 5.75 times in 2015. The impact factor undergoes natural variation according to citation rates of papers in the 2 years following publication, and is significantly influenced by highly cited papers such as official reports. However, the progress of the journal's impact over the last 5 years has been impressive. Our acceptance rate is < 25 % and has been falling because the number of articles being submitted has been increasing. In accordance with Open-Access publishing, the JCMR articles go on-line as they are accepted with no collating of the articles into sections or special thematic issues. For this reason, the Editors have felt that it is useful once per calendar year to summarize the papers for the readership into broad areas of interest or theme, so that areas of interest can be reviewed in a single article in relation to each other and other recent JCMR articles. The papers are presented in broad themes and set in context with related literature and previously published JCMR papers to guide continuity of thought in the journal. We hope that you find the open-access system increases wider reading and citation of your papers, and that you will continue to send your quality papers to JCMR for publication.
rea G, Homfray T, Till J, et al., 2016, Histiocytoid cardiomyopathy and microphthalmia with linear skin defects syndrome: phenotypes linked by truncating variants in NDUFB11, Cold Spring Harbor Molecular Case Studies, Vol: 3, ISSN: 2373-2873
Variants in NDUFB11, which encodes a structural component of complex I of the mitochondrial respiratory chain (MRC), were recently independently reported to cause histiocytoid cardiomyopathy (histiocytoid CM) and microphthalmia with linear skin defects syndrome (MLS syndrome). Here we report an additional case of histiocytoid CM, which carries a de novo nonsense variant in NDUFB11 (ENST00000276062.8: c.262C > T; p.[Arg88*]) identified using whole-exome sequencing (WES) of a family trio. An identical variant has been previously reported in association with MLS syndrome. The case we describe here lacked the diagnostic features of MLS syndrome, but a detailed clinical comparison of the two cases revealed significant phenotypic overlap. Heterozygous variants in HCCS (which encodes an important mitochondrially targeted protein) and COX7B, which, like NDUFB11, encodes a protein of the MRC, have also previously been identified in MLS syndrome including a case with features of both MLS syndrome and histiocytoid CM. However, a systematic review of WES data from previously published histiocytoid CM cases, alongside four additional cases presented here for the first time, did not identify any variants in these genes. We conclude that NDUFB11 variants play a role in the pathogenesis of both histiocytoid CM and MLS and that these disorders are allelic (genetically related).
Izgi C, Vassiliou V, Baksi AJ, et al., 2016, Differential diagnosis of left ventricular hypertrophy: usefulness of multimodality imaging and tissue characterization with cardiac magnetic resonance., Echocardiography, ISSN: 0742-2822
Differential diagnosis of asymmetrical left ventricular hypertrophy may be challenging, particularly in patients with history of hypertension. A middle-aged man underwent an echocardiographic examination during workup for hypertension, which unexpectedly showed significant asymmetrical septal hypertrophy and raised suspicion for hypertrophic cardiomyopathy. Cardiovascular magnetic resonance confirmed the asymmetrical hypertrophy. No myocardial late gadolinium contrast enhancement was seen. However, precontrast T1 mapping revealed a low native myocardial T1 value. This was highly suggestive of Anderson-Fabry disease, which was subsequently proved with very low alpha galactosidase enzyme levels and mutation analysis. The case illustrates clinical usefulness of multimodality imaging and the novel tissue characterization techniques for assessment of left ventricular hypertrophy.
Alam MH, Auger D, McGill LA, et al., 2016, Comparison of 3 T and 1.5 T for T2* magnetic resonance of tissue iron, Journal of Cardiovascular Magnetic Resonance, Vol: 18, ISSN: 1532-429X
BACKGROUND: T2* magnetic resonance of tissue iron concentration has improved the outcome of transfusion dependant anaemia patients. Clinical evaluation is performed at 1.5 T but scanners operating at 3 T are increasing in numbers. There is a paucity of data on the relative merits of iron quantification at 3 T vs 1.5 T. METHODS: A total of 104 transfusion dependent anaemia patients and 20 normal volunteers were prospectively recruited to undergo cardiac and liver T2* assessment at both 1.5 T and 3 T. Intra-observer, inter-observer and inter-study reproducibility analysis were performed on 20 randomly selected patients for cardiac and liver T2*. RESULTS: Association between heart and liver T2* at 1.5 T and 3 T was non-linear with good fit (R (2) = 0.954, p < 0.001 for heart white-blood (WB) imaging; R (2) = 0.931, p < 0.001 for heart black-blood (BB) imaging; R (2) = 0.993, p < 0.001 for liver imaging). R2* approximately doubled between 1.5 T and 3 T with linear fits for both heart and liver (94, 94 and 105 % respectively). Coefficients of variation for intra- and inter-observer reproducibility, as well as inter-study reproducibility trended to be less good at 3 T (3.5 to 6.5 %) than at 1.5 T (1.4 to 5.7 %) for both heart and liver T2*. Artefact scores for the heart were significantly worse with the 3 T BB sequence (median 4, IQR 2-5) compared with the 1.5 T BB sequence (4 [3-5], p = 0.007). CONCLUSION: Heart and liver T2* and R2* at 3 T show close association with 1.5 T values, but there were more artefacts at 3 T and trends to lower reproducibility causing difficulty in quantifying low T2* values with high tissue iron. Therefore T2* imaging at 1.5 T remains the gold standard for clinical practice. However, in centres where only 3 T is available, equival
Rea G, Homfray T, Till J, et al., 2016, Whole Exome Sequencing Identifies Genetic Cause of Histiocytoid Cardiomyopathy, Annual Conference of the British-Cardiovascular-Society (BCS) on Prediction and Prevention, Publisher: BMJ Publishing Group, Pages: A138-A139, ISSN: 1468-201X
Histiocytoid cardiomyopathy (CM) is a rare, distinctive form of cardiomyopathy, characterised by malignant arrhythmias and associated sudden death. ~90% of cases present in females <2 years of age. We undertook whole exome sequencing (WES) in five unrelated affected females, including one with parental samples available. In the family trio we identified a de novo nonsense mutation in NDUFB11 (c.262C>T; p. Arg88*), located on the X chromosome and encoding a component of the mitochondrial respiratory chain (MRC). Mutations in NDUFB11, including one identical to the one we describe here, have been reported to cause microphthalmia and linear skin defects syndrome (MLS). During the course of our studies, additional mutations in NDUFB11 were associated with Histiocytoid CM by another group. Four of the affected individuals in our study did not carry variants in NDUFB11. Heterozygous mutations in HCCS (which encodes an important mitochondrially-targeted protein) and COX7B (which, like NDUFB11, encodes a protein of the MRC) have also previously been identified in MLS syndrome including a case with features of both MLS syndrome and Histiocytoid CM. However, a systematic review of WES data from previously published cases, alongside the four additional cases presented here, did not identify any further mutations in these genes in Histiocytoid CM. We conclude thattruncating variants in NDUFB11 link the distinct phenotypes of Histiocytoid CM and MLS syndrome. Screening for malignant arrhythmias and cardiomyopathy would be appropriate in individuals with MLS syndrome. Additional nuclear encoded mitochondrial or mitochondrial DNA genes are good candidates for further causes of both Histiocytoid CM and MLS syndrome.
Rea G, Ware JS, Homfray T, et al., 2016, HISTIOCYTOID CARDIOMYOPATHY AND MICROPHTHALMIA WITH LINEAR SKIN DEFECTS SYNDROME: PHENOTYPES LINKED BY TRUNCATING VARIANTS IN <i>NDUFB11</i>, Annual Meeting of the British-Congenital-Cardiac-Association, Publisher: BMJ PUBLISHING GROUP, Pages: A18-A18, ISSN: 1355-6037
Alam MH, Auger D, Smith GC, et al., 2015, T1 at 1.5T and 3T compared with conventional T2*at 1.5T for cardiac siderosis, Journal of Cardiovascular Magnetic Resonance, Vol: 17, ISSN: 1532-429X
Background: Myocardial black blood (BB) T2* relaxometry at 1.5T provides robust, reproducible and calibratednon-invasive assessment of cardiac iron burden. In vitro data has shown that like T2*, novel native ModifiedLook-Locker Inversion recovery (MOLLI) T1 shortens with increasing tissue iron. The relative merits of T1 andT2* are largely unexplored. We compared the established 1.5T BB T2* technique against native T1 values at1.5T and 3T in iron overload patients and in normal volunteers.Methods: A total of 73 subjects (42 male) were recruited, comprising 20 healthy volunteers (controls) and 53 patients(thalassemia major 22, sickle cell disease 9, hereditary hemochromatosis 9, other iron overload conditions 13). Singlemid-ventricular short axis slices were acquired for BB T2* at 1.5T and MOLLI T1 quantification at 1.5T and 3T.Results: In healthy volunteers, median T1 was 1014 ms (full range 939–1059 ms) at 1.5T and modestly increased to1165ms (full range 1056–1224 ms) at 3T. All patients with significant cardiac iron overload (1.5T T2* values <20 ms) hadT1 values <939 ms at 1.5T, and <1056 ms at 3T. Associations between T2* and T1 were found to be moderatewith y =377 · x0.282 at 1.5T (R2 = 0.717), and y =406 · x0.294 at 3T (R2 = 0.715). Measures of reproducibility of T1appeared superior to T2*.Conclusions: T1 mapping at 1.5T and at 3T can identify individuals with significant iron loading as defined bythe current gold standard T2* at 1.5T. However, there is significant scatter between results which may reflectmeasurement error, but it is also possible that T1 interacts with T2*, or is differentially sensitive to aspects of ironchemistry or other biology. Hurdles to clinical implementation of T1 include the lack of calibration against humanmyocardial iron concentration, no demonstrated relation to cardiac outcomes, and variation in absolute T1 valuesbetween scanners, which makes inter-centre comparisons difficult. The relative merit
Pennell DJ, Baksi AJ, Prasad SK, et al., 2015, Review of Journal of Cardiovascular Magnetic Resonance 2014, Journal of Cardiovascular Magnetic Resonance, Vol: 17, ISSN: 1532-429X
There were 102 articles published in the Journal of Cardiovascular Magnetic Resonance (JCMR) in 2014, which is a6 % decrease on the 109 articles published in 2013. The quality of the submissions continues to increase. The 2013JCMR Impact Factor (which is published in June 2014) fell to 4.72 from 5.11 for 2012 (as published in June 2013).The 2013 impact factor means that the JCMR papers that were published in 2011 and 2012 were cited on average4.72 times in 2013. The impact factor undergoes natural variation according to citation rates of papers in the 2 yearsfollowing publication, and is significantly influenced by highly cited papers such as official reports. However,the progress of the journal’s impact over the last 5 years has been impressive. Our acceptance rate is <25 %and has been falling because the number of articles being submitted has been increasing. In accordance withOpen-Access publishing, the JCMR articles go on-line as they are accepted with no collating of the articlesinto sections or special thematic issues. For this reason, the Editors have felt that it is useful once per calendar year tosummarize the papers for the readership into broad areas of interest or theme, so that areas of interest canbe reviewed in a single article in relation to each other and other recent JCMR articles. The papers are presented inbroad themes and set in context with related literature and previously published JCMR papers to guide continuity ofthought in the journal. We hope that you find the open-access system increases wider reading and citation of yourpapers, and that you will continue to send your quality papers to JCMR for publication.
Baksi AJ, Davies JE, Hadjiloizou N, et al., 2015, Attenuation of reflected waves in man during retrograde propagation from femoral artery to proximal aorta, International Journal of Cardiology, Vol: 202, Pages: 441-445, ISSN: 1874-1754
BackgroundWave reflection may be an important influence on blood pressure, but the extent to which reflections undergo attenuation during retrograde propagation has not been studied. We quantified retrograde transmission of a reflected wave created by occlusion of the left femoral artery in man.Methods20 subjects (age 31–83 years; 14 male) underwent invasive measurement of pressure and flow velocity with a sensor-tipped intra-arterial wire at multiple locations distal to the proximal aorta before, during and following occlusion of the left femoral artery by thigh cuff inflation. A numerical model of the circulation was also used to predict reflected wave transmission. Wave reflection was measured as the ratio of backward to forward wave energy (WRI) and the ratio of peak backward to forward pressure (Pb/Pf).ResultsCuff inflation caused a marked reflection which was largest at 5–10 cm from the cuff (change (Δ) in WRI = 0.50 (95% CI 0.38, 0.62); p < 0.001, ΔPb/Pf = 0.23 (0.18–0.29); p < 0.001). The magnitude of the cuff-induced reflection decreased progressively at more proximal locations and was barely discernible at sites > 40 cm from the cuff including in the proximal aorta. Numerical modelling gave similar predictions to those observed experimentally.ConclusionsReflections due to femoral artery occlusion are markedly attenuated by the time they reach the proximal aorta. This is due to impedance mismatches of bifurcations traversed in the backward direction. This degree of attenuation is inconsistent with the idea of a large discrete reflected wave arising from the lower limb and propagating back into the aorta.
Baksi AJ, Pennell DJ, 2015, Cardiomyopathy, Advanced Cardiac Imaging, Pages: 399-438, ISBN: 9781782422822
Izgi C, Ray S, Nyktari E, et al., 2015, Myocardial edema in Takotsubo syndrome mimicking apical hypertrophic cardiomyopathy: An insight into diagnosis by cardiovascular magnetic resonance, Heart & Lung, Vol: 44, Pages: 481-485, ISSN: 0147-9563
Myocardial edema is one of the characteristic features in the pathogenesis of Takotsubo syndrome. We report a middle aged man who presented with typical clinical and echocardiographic features of apical variant of Takotsubo syndrome. However, a cardiovascular magnetic resonance study performed 10 days after presentation did not show any apical ‘ballooning’ but revealed features of an apical hypertrophic cardiomyopathy on cine images. Tissue characterization with T2 weighted images proved severe edema as the cause of significantly increased apical wall thickness. A follow-up cardiovascular magnetic resonance study was performed 5 months later which showed that edema, wall thickening and the appearance of apical hypertrophic cardiomyopathy all resolved, confirming Takotsubo syndrome as the cause of the initial appearance. As the affected myocardium most commonly involves the apical segments, an edema induced increase in apical wall thickness may lead to appearances of an apical hypertrophic cardiomyopathy rather than apical ballooning in the acute to subacute phase of Takotsubo syndrome.
Venneri L, Calicchio F, Manivarmane R, et al., 2015, Impaired myocardial function despite normal ejection fraction in patients on current cancer drug therapies, Congress of the European-Society-of-Cardiology (ESC), Publisher: OXFORD UNIV PRESS, Pages: 150-150, ISSN: 0195-668X
Baksi AJ, Kanaganayagam GS, Prasad SK, 2015, Arrhythmias in viral myocarditis and pericarditis., Card Electrophysiol Clin, Vol: 7, Pages: 269-281
Acute viral myocarditis and acute pericarditis are self-limiting conditions that run a benign course and that may not involve symptoms that lead to medical assessment. However, ventricular arrhythmia is frequent in viral myocarditis. Myocarditis is thought to account for a large proportion of sudden cardiac deaths in young people without prior structural heart disease. Identification of acute myocarditis either with or without pericarditis is therefore important. However, therapeutic interventions are limited and nonspecific. Identifying those at greatest risk of a life-threatening arrhythmia is critical to reducing the mortality. This review summarizes current understanding of this challenging area in which many questions remain.
Pennell DJ, Baksi AJ, Kilner PJ, et al., 2014, Review of Journal of Cardiovascular Magnetic Resonance 2013, Journal of Cardiovascular Magnetic Resonance, Vol: 16, ISSN: 1532-429X
There were 109 articles published in the Journal of Cardiovascular Magnetic Resonance (JCMR) in 2013, which is a21% increase on the 90 articles published in 2012. The quality of the submissions continues to increase. The editorsare delighted to report that the 2012 JCMR Impact Factor (which is published in June 2013) has risen to 5.11, upfrom 4.44 for 2011 (as published in June 2012), a 15% increase and taking us through the 5 threshold for the firsttime. The 2012 impact factor means that the JCMR papers that were published in 2010 and 2011 were cited onaverage 5.11 times in 2012. The impact factor undergoes natural variation according to citation rates of papers inthe 2 years following publication, and is significantly influenced by highly cited papers such as official reports.However, the progress of the journal's impact over the last 5 years has been impressive. Our acceptance rate is<25% and has been falling because the number of articles being submitted has been increasing. In accordancewith Open-Access publishing, the JCMR articles go on-line as they are accepted with no collating of the articlesinto sections or special thematic issues. For this reason, the Editors have felt that it is useful once per calendaryear to summarize the papers for the readership into broad areas of interest or theme, so that areas of interestcan be reviewed in a single article in relation to each other and other recent JCMR articles. The papers arepresented in broad themes and set in context with related literature and previously published JCMR papers toguide continuity of thought in the journal. We hope that you find the open-access system increases widerreading and citation of your papers, and that you will continue to send your quality manuscripts to JCMR forpublication.
Baksi AJ, Pennell DJ, 2014, Randomized controlled trials of iron chelators for the treatment of cardiac siderosis in thalassaemia major, FRONTIERS IN PHARMACOLOGY, Vol: 5, ISSN: 1663-9812
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- Citations: 26
Raphael CE, Baksi AJ, Alpendurada F, et al., 2014, Cardiovascular magnetic resonance in patients with MRI-conditional pacemakers: a single centre experience, Annual Meeting of the European-Society-of-Cardiology (ESC), Publisher: OXFORD UNIV PRESS, Pages: 798-799, ISSN: 0195-668X
Carpenter J-P, He T, Kirk P, et al., 2014, Calibration of myocardial T2 and T1 against iron concentration, JOURNAL OF CARDIOVASCULAR MAGNETIC RESONANCE, Vol: 16, ISSN: 1097-6647
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- Citations: 28
Petryka J, Baksi AJ, Prasad SK, et al., 2014, Prevalence of Inferobasal Myocardial Crypts Among Patients Referred for Cardiovascular Magnetic Resonance, CIRCULATION-CARDIOVASCULAR IMAGING, Vol: 7, Pages: 259-264, ISSN: 1941-9651
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- Citations: 28
Baksi AJ, Pennell DJ, 2014, T2* imaging of the heart: methods, applications, and outcomes., Top Magn Reson Imaging, Vol: 23, Pages: 13-20
This review describes and discusses the rationale, technique, applications, and impact of cardiovascular magnetic resonance (CMR) T2* imaging, principally in the assessment of iron loading within the heart, and highlights how this robust imaging strategy has transformed disease outcome.Until recently, no simple noninvasive measurement was available to reliably indicate severe cardiac iron loading before the development of overt cardiac dysfunction or heart failure. Consequently, the majority of patients with transfusion-dependent anemias, such as β-thalassemia major, died prematurely of cardiovascular complications of severe iron overload.The magnetic properties of particulate iron disrupt magnetic field homogeneity in the CMR environment and consequently influence the CMR parameter T2*, which describes signal decay relating to both field inhomogeneity and loss of spin coherence. There is a direct relationship between T2* and myocardial iron concentration, enabling this to be used to identify and quantify myocardial iron load. Single breath-hold gradient-echo sequences in which a single midventricular short-axis myocardial slice is acquired at multiple echo times enables a myocardial T2* value to be measured from the rate of exponential decay. The application of T2* CMR to assessing cardiac iron loading is rapid, reproducible, extensively validated, and now widely performed. Data have highlighted the profound predictive power of this imaging technique and moreover its ability to inform management strategies such that, over a relatively short duration, outcome has been dramatically improved, and the disease course in β-thalassemia major transformed.
Barron AJ, Hughes AD, Sharp A, et al., 2014, Long-Term Antihypertensive Treatment Fails to Improve E/e′ Despite Regression of Left Ventricular Mass An Anglo-Scandinavian Cardiac Outcomes Trial Substudy, HYPERTENSION, Vol: 63, Pages: 252-+, ISSN: 0194-911X
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- Citations: 20
Baksi AJ, Pennell DJ, 2013, T1 Mapping in Heart Failure From Technique to Prognosis, Toward Altering Outcome, CIRCULATION-CARDIOVASCULAR IMAGING, Vol: 6, Pages: E58-E60, ISSN: 1941-9651
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- Citations: 8
Pennell DJ, Baksi AJ, Carpenter JP, et al., 2013, Review of Journal of Cardiovascular Magnetic Resonance 2012, JOURNAL OF CARDIOVASCULAR MAGNETIC RESONANCE, Vol: 15, ISSN: 1097-6647
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- Citations: 5
Gulati A, Ismail TF, Jabbour A, et al., 2013, Clinical utility and prognostic value of left atrial volume assessment by cardiovascular magnetic resonance in non-ischaemic dilated cardiomyopathy, EUROPEAN JOURNAL OF HEART FAILURE, Vol: 15, Pages: 660-670, ISSN: 1388-9842
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- Citations: 79
O'Regan DP, Ariff B, Baksi AJ, et al., 2013, Salvage assessment with cardiac MRI following acute myocardial infarction underestimates potential for recovery of systolic strain, EUROPEAN RADIOLOGY, Vol: 23, Pages: 1210-1217, ISSN: 0938-7994
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- Citations: 11
Gulati A, Ismail N, Ismail T, et al., 2012, Left Atrial Volume Provides Independent and Incremental Prognostic Information in Non-Ischemic Dilated Cardiomyopathy, CIRCULATION, Vol: 126, ISSN: 0009-7322
O'Regan DP, Shi W, Ariff B, et al., 2012, Remodeling after acute myocardial infarction: mapping ventricular dilatation using three dimensional CMR image registration, Journal of Cardiovascular Magnetic Resonance, Vol: 14, ISSN: 1532-429X
Background: Progressive heart failure due to remodeling is a major cause of morbidity and mortality followingmyocardial infarction. Conventional clinical imaging measures global volume changes, and currently there is nomeans of assessing regional myocardial dilatation in relation to ischemic burden. Here we use 3D co-registration ofCardiovascular Magnetic Resonance (CMR) images to assess the long-term effects of ischemia-reperfusion injury onleft ventricular structure after acute ST-elevation myocardial infarction (STEMI).Methods: Forty six patients (age range 33–77 years) underwent CMR imaging within 7 days following primarypercutaneous coronary intervention (PPCI) for acute STEMI with follow-up at one year. Functional cine imaging andLate Gadolinium Enhancement (LGE) were segmented and co-registered. Local left ventricular wall dilatation wasassessed by using intensity-based similarities to track the structural changes in the heart between baseline andfollow-up. Results are expressed as means, standard errors and 95% confidence interval (CI) of the difference.Results: Local left ventricular remodeling within infarcted myocardium was greater than in non-infarctedmyocardium (1.6% ± 1.0 vs 0.3% ± 0.9, 95% CI: -2.4% – -0.2%, P = 0.02). One-way ANOVA revealed that transmuralinfarct thickness had a significant effect on the degree of local remodeling at one year (P < 0.0001) with greatestwall dilatation observed when infarct transmurality exceeded 50%. Infarct remodeling was more severe whenmicrovascular obstruction (MVO) was present (3.8% ± 1.3 vs −1.6% ± 1.4, 95% CI: -9.1% – -1.5%, P = 0.007) and whenend-diastolic volume had increased by >20% (4.8% ± 1.4 vs −0.15% ± 1.2, 95% CI: -8.9% – -0.9%, P = 0.017).Conclusions: The severity of ischemic injury has a significant effect on local ventricular wall remodeling with onlymodest dilatation observed within non-ischemic myocardium. Limitatio
Barron AJ, Tapp RJ, Sharp A, et al., 2011, Long-Term Differential Effects of Amlodipine and Atenolol based Antihypertensive Therapy on Left Ventricular Structure and Function: An ASCOT Sub-Study, CIRCULATION, Vol: 124, ISSN: 0009-7322
Baksi AJ, Davies JE, Hadjiloizou N, et al., 2011, Backward Propagation of Waves is Limited in the Human Arterial Tree, CIRCULATION, Vol: 124, ISSN: 0009-7322
Baksi AJ, 2011, Common academic pitfalls., Secrets of Success: Getting into Academic Medicine., Editors: Smith, Gill, Nijjer, Levy, London, Publisher: RSM Books; Hodder Arnold, ISBN: 978-1853159572
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