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@article{Dimakopoulou:2019:10.3389/fendo.2019.00628,
author = {Dimakopoulou, A and Jayasena, CN and Radia, UK and Algefari, M and Minhas, S and Oliver, N and Dhillo, WS},
doi = {10.3389/fendo.2019.00628},
journal = {Frontiers in Endocrinology},
title = {Animal models of diabetes-related male hypogonadism},
url = {http://dx.doi.org/10.3389/fendo.2019.00628},
volume = {10},
year = {2019}
}

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TY  - JOUR
AB  - Hypogonadism is the clinical syndrome associated with low testosterone secretion in men. Hypogonadism affects ~37–57% men with diabetes mellitus (1). Male reproduction is orchestrated by the hypothalamo-pituitary-gonadal (HPG) axis, which regulates the biosynthesis of testosterone from the testes. Diabetes may cause hypogonadism through multiple mechanisms including suppression of hypothalamic gonadotrophin-releasing hormone (GnRH) secretion, or direct disruption of spermatogenesis (2). Clinical stigmata of hypogonadism include reduced libido, erectile dysfunction (ED) and reduced physical strength. This article will summarize the evidence from animal models including how diabetes affects male reproductive endocrine function and predisposes to hypogonadism.
AU  - Dimakopoulou,A
AU  - Jayasena,CN
AU  - Radia,UK
AU  - Algefari,M
AU  - Minhas,S
AU  - Oliver,N
AU  - Dhillo,WS
DO  - 10.3389/fendo.2019.00628
PY  - 2019///
SN  - 1664-2392
TI  - Animal models of diabetes-related male hypogonadism
T2  - Frontiers in Endocrinology
UR  - http://dx.doi.org/10.3389/fendo.2019.00628
UR  - http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000486529400001&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=1ba7043ffcc86c417c072aa74d649202
UR  - http://hdl.handle.net/10044/1/74835
VL  - 10
ER  -

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