19 results found
Jenkins PO, De Simoni S, Bourke NJ, et al., 2018, Dopaminergic abnormalities following traumatic brain injury, BRAIN, Vol: 141, Pages: 797-810, ISSN: 0006-8950
Scott G, Zetterberg H, Jolly A, et al., 2018, Minocycline reduces chronic microglial activation after brain trauma but increases neurodegeneration, BRAIN, Vol: 141, Pages: 459-471, ISSN: 0006-8950
De Simoni S, Jenkins PO, Bourke NJ, et al., 2018, Altered caudate connectivity is associated with executive dysfunction after traumatic brain injury, BRAIN, Vol: 141, Pages: 148-164, ISSN: 0006-8950
Feeney C, Sharp DJ, Hellyer PJ, et al., 2017, Serum Insulin-like Growth Factor-I Levels are Associated with Improved White Matter Recovery after Traumatic Brain Injury, ANNALS OF NEUROLOGY, Vol: 82, Pages: 30-43, ISSN: 0364-5134
Scott G, Jolly A, Jenkins PO, et al., 2016, THE EFFECT OF MINOCYCLINE ON NEUROINFLAMMATION AFTER BRAIN TRAUMA, Annual Meeting of the Association-of-British-Neurologists (ABN), Publisher: BMJ PUBLISHING GROUP, ISSN: 0022-3050
Jenkins PO, De Simoni S, Fleminger J, et al., 2016, DISRUPTION TO THE DOPAMINERGIC SYSTEM AFTER TRAUMATIC BRAIN INJURY, Annual Meeting of the Association-of-British-Neurologists (ABN), Publisher: BMJ PUBLISHING GROUP, ISSN: 0022-3050
De Simoni S, Grover PJ, Jenkins PO, et al., 2016, Disconnection between the default mode network and medial temporal lobes in post-traumatic amnesia, BRAIN, Vol: 139, Pages: 3137-3150, ISSN: 0006-8950
Jamall OA, Feeney C, Zaw-Linn J, et al., 2016, Prevalence and correlates of vitamin D deficiency in adults after traumatic brain injury, CLINICAL ENDOCRINOLOGY, Vol: 85, Pages: 636-644, ISSN: 0300-0664
Jenkins PO, Mehta MA, Sharp DJ, 2016, Catecholamines and cognition after traumatic brain injury, BRAIN, Vol: 139, Pages: 2345-2371, ISSN: 0006-8950
Jenkins P, De Simoni S, Fleminger J, et al., 2016, Disruption to the dopaminergic system following traumatic brain injury, Publisher: TAYLOR & FRANCIS INC, Pages: 670-670, ISSN: 0269-9052
Sharp DJ, Jenkins PO, 2015, Concussion is confusing us all., Pract Neurol, Vol: 15, Pages: 172-186
It is time to stop using the term concussion as it has no clear definition and no pathological meaning. This confusion is increasingly problematic as the management of 'concussed' individuals is a pressing concern. Historically, it has been used to describe patients briefly disabled following a head injury, with the assumption that this was due to a transient disorder of brain function without long-term sequelae. However, the symptoms of concussion are highly variable in duration, and can persist for many years with no reliable early predictors of outcome. Using vague terminology for post-traumatic problems leads to misconceptions and biases in the diagnostic process, producing uninterpretable science, poor clinical guidelines and confused policy. We propose that the term concussion should be avoided. Instead neurologists and other healthcare professionals should classify the severity of traumatic brain injury and then attempt to precisely diagnose the underlying cause of post-traumatic symptoms.
Bantel H, Thum T, Schulze-Osthoff K, 2015, Understanding the Pathophysiological Regulatory Role of MicroRNAs in Acute Liver Failure Reply, HEPATOLOGY, Vol: 61, Pages: 1440-1441, ISSN: 0270-9139
Jenkins PO, Perry R, Malik O, 2014, Multiple sclerosis presenting as a relapsing amnestic syndrome., Pract Neurol, Vol: 14, Pages: 100-101
Jenkins PO, Soper C, MacKinnon AD, et al., 2014, Systemic Lupus Erythematosus Presenting as Orbital Myositis., Neuroophthalmology, Vol: 38, Pages: 264-267, ISSN: 0165-8107
The authors present a case of diplopia and eye pain due to orbital myositis in a patient with a de novo diagnosis of systemic lupus erythematosus. Systemic lupus erythematosus is a rare cause of orbital myositis and should be considered when other, more common, conditions have been excluded.
Reitboeck PG, Jenkins P, Pereira A, et al., 2013, STARRY NIGHTS: COMA DUE TO CEREBRAL FAT EMBOLISM SYNDROME, JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY, Vol: 84, ISSN: 0022-3050
, 2010, In response, Clinical Medicine, Journal of the Royal College of Physicians of London, Vol: 10, ISSN: 1470-2118
Jenkins PO, Sultanzadeh J, Bhagwat M, et al., 2009, Should thrombolysis have a greater role in the management of pulmonary embolism?, CLINICAL MEDICINE, Vol: 9, Pages: 431-435, ISSN: 1470-2118
, 2009, Should thrombolysis have a greater role in the management of pulmonary embolism?, Clinical Medicine, Journal of the Royal College of Physicians of London, Vol: 9, Pages: 432-435, ISSN: 1470-2118
Pulmonary embolism (PE) continues to be associated with significant mortality despite advances in the diagnostic techniques available for its detection. Anticoagulation remains standard treatment in PE although there is a consensus view that 'step-up' to thrombolytic therapy in addition to anticoagulation is indicated in those patients who are systemically shocked at presentation - a group defined as having suffered 'massive pulmonary embolism'. Considerable research has been directed at attempting to identify further groups of patients with PE who are at high risk of morbidity and mortality - notably those who are labelled as having suffered 'sub-massive pulmonary embolism' where this is defined as the presence of right-heart strain in the absence of systemic shock. In particular, the potential benefit of extending thrombolytic therapy to include those patients with sub-massive PE has been the subject of much enquiry and debate. This review examines the evidence for thrombolytic therapy and explores the potential for risk stratification in PE. © Royal College of Physicians, 2009. All rights reserved.
Jenkins PO, Turner MR, Jenkins PF, 2008, What is the place of thrombolysis in acute stroke? A review of the literature and a current perspective, CLINICAL MEDICINE, Vol: 8, Pages: 253-258, ISSN: 1470-2118
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