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• Book chapter
  Song B, Bismarck A, Springer J, 2004,

  Contact angle measurements on fibers and fiber assemblies, bundles, fabrics and textiles

  , Surface and interfacial tension: measurement, theory, and applications, Editors: Hartland, New York, Publisher: Marcel Dekker, Pages: 425-481, ISBN: 9780824750343
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• Book chapter
  Arst HN Jr, Tilburn J, 2004,

  Regulation of gene expression by ambient pH.

  , The Mycota Vol. III. Biochemistry and Molecular Biology., Editors: Brambl, Marzluf, Brambl, Marzluf, Brambl, Marzluf, Berlin, Publisher: Springer-Verlag, Pages: 121-128, ISBN: 9783540426301
  • Cite
• Conference paper
  Horsefield R, Yankovskaya V, Byrne B, Cecchini G, Iwata Set al., 2004,

  The structure of complex II and its quinone-binding site

  , 13th European Bioenergetics Conference (EBEC 2004), Publisher: ELSEVIER SCIENCE BV, Pages: 165-165, ISSN: 0005-2728
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• Journal article
  Adcock IM, Ito K, Barnes PJ, 2004,

  Glucocorticoids: effects on gene transcription.

  , Proc Am Thorac Soc, Vol: 1, Pages: 247-254, ISSN: 1546-3222

  The major antiinflammatory effects of glucocorticoids appear to be due largely to interaction between the activated glucocorticoid receptor and transcription factors, notably nuclear factor-kappaB (NF-kappaB) and activator protein-1, that mediate the expression of inflammatory genes. NF-kappaB switches on inflammatory genes via a process involving recruitment of transcriptional coactivator proteins and changes in chromatin modifications such as histone acetylation. This process must occur in the correct temporal manner to allow for effective inflammatory gene expression to occur. Glucocorticoids, using a similar mechanism, are also able to switch on a number of antiinflammatory genes. An important question is why glucocorticoids switch off only inflammatory genes, as they clearly do not suppress all activated genes and are well tolerated as long-term treatments. The interactions between NF-kappaB and the glucocorticoid receptor result in differing effects on histone acetylation and deacetylation. Oxidative stress due to cigarette smoke may be an important factor in inducing glucocorticoid resistance in chronic obstructive pulmonary disease and may involve changes in histone acetylation/deacetylation balance.

  • Abstract
  • Author Web Link
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• Book chapter
  Both M, 2004,

  Blumeria graminis f. sp. hordei, an obligate pathogen of barle

  , Plant Pathogen Interactions, Editors: Talbot, Oxford, Publisher: Blackwell publishing, Pages: 202-218
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• Journal article
  Cookson W, Moffatt M, 2004,

  Making sense of asthma genes.

  , N Engl J Med, Vol: 351, Pages: 1794-1796
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• Journal article
  Peñalva MA, Arst HN, 2004,

  Recent advances in the characterization of ambient pH regulation of gene expression in filamentour fungi and yeasts

  , ANNUAL REVIEW OF MICROBIOLOGY, Vol: 58, Pages: 425-451, ISSN: 0066-4227
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  • Citations: 158
• Journal article
  Bismarck A, Schulz E, 2003,

  Adhesion and friction behavior between fluorinated carbon fibers and poly (vinylidene fluoride)

  , JOURNAL OF MATERIALS SCIENCE, Vol: 38, Pages: 4965-4972, ISSN: 0022-2461
  • Author Web Link
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  • Citations: 31
• Journal article
  Fernández-Martínez J, Brown CV, Díez E, Tilburn J, Arst HN, Peñalva MA, Espeso EAet al., 2003,

  Overlap of nuclear localisation signal and specific DNA-binding residues within the zinc finger domain of PacC

  , JOURNAL OF MOLECULAR BIOLOGY, Vol: 334, Pages: 667-684, ISSN: 0022-2836
  • Author Web Link
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  • Citations: 65
• Journal article
  Cookson W, 2003,

  July 1957 (from the trifler) section: Rock -Drill

  , Agenda, Vol: 39, ISSN: 0002-0796
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