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  • Conference paper
    Fears MA, Dumontet V, Okubanjo O, Bismarck A, Sakuri T, Pontzen Ret al., 2004,

    Effect of formulation components on the bulk stability and foliar delivery behaviour of suspo-emulsion formulations containing built-in adjuvants

    , 7th international symposium on adjuvants for agrochemicals (ISAA2004), Cape Town, SA, 8 - 12 November 2004
  • Book chapter
    Song B, Bismarck A, Springer J, 2004,

    Contact angle measurements on fibers and fiber assemblies, bundles, fabrics and textiles

    , Surface and interfacial tension: measurement, theory, and applications, Editors: Hartland, New York, Publisher: Marcel Dekker, Pages: 425-481, ISBN: 9780824750343
  • Journal article
    Aziz ZA, Wells AU, Hansell DM, Bain GA, Copley SJet al., 2004,

    HRCT diagnosis of diffuse parenchymal lung disease: inter-observer variation

    , Thorax
  • Book chapter
    Arst HN Jr, Tilburn J, 2004,

    Regulation of gene expression by ambient pH.

    , The Mycota Vol. III. Biochemistry and Molecular Biology., Editors: Brambl, Marzluf, Brambl, Marzluf, Brambl, Marzluf, Berlin, Publisher: Springer-Verlag, Pages: 121-128, ISBN: 9783540426301
  • Book chapter
    Both M, 2004,

    Blumeria graminis f. sp. hordei, an obligate pathogen of barle

    , Plant Pathogen Interactions, Editors: Talbot, Oxford, Publisher: Blackwell publishing, Pages: 202-218
  • Journal article
    Davies JC, 2004,

    Modifier genes in cystic fibrosis

    , PEDIATRIC PULMONOLOGY, Pages: 86-87, ISSN: 8755-6863
  • Journal article
    Cookson W, Moffatt M, 2004,

    Making sense of asthma genes.

    , N Engl J Med, Vol: 351, Pages: 1794-1796
  • Journal article
    Peñalva MA, Arst HN, 2004,

    Recent advances in the characterization of ambient pH regulation of gene expression in filamentour fungi and yeasts

    , ANNUAL REVIEW OF MICROBIOLOGY, Vol: 58, Pages: 425-451, ISSN: 0066-4227
    • Cite
    • Citations: 164
  • Journal article
    Adcock IM, Ito K, Barnes PJ, 2004,

    Glucocorticoids: effects on gene transcription.

    , Proc Am Thorac Soc, Vol: 1, Pages: 247-254, ISSN: 1546-3222

    The major antiinflammatory effects of glucocorticoids appear to be due largely to interaction between the activated glucocorticoid receptor and transcription factors, notably nuclear factor-kappaB (NF-kappaB) and activator protein-1, that mediate the expression of inflammatory genes. NF-kappaB switches on inflammatory genes via a process involving recruitment of transcriptional coactivator proteins and changes in chromatin modifications such as histone acetylation. This process must occur in the correct temporal manner to allow for effective inflammatory gene expression to occur. Glucocorticoids, using a similar mechanism, are also able to switch on a number of antiinflammatory genes. An important question is why glucocorticoids switch off only inflammatory genes, as they clearly do not suppress all activated genes and are well tolerated as long-term treatments. The interactions between NF-kappaB and the glucocorticoid receptor result in differing effects on histone acetylation and deacetylation. Oxidative stress due to cigarette smoke may be an important factor in inducing glucocorticoid resistance in chronic obstructive pulmonary disease and may involve changes in histone acetylation/deacetylation balance.
  • Journal article
    Davies JC, 2004,

    Ion transport in lung disease

    , PEDIATRIC PULMONOLOGY, Pages: 147-148, ISSN: 8755-6863

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