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  • Journal article
    Vincent O, Rainbow L, Tilburn J, Arst HN, Peñalva MAet al., 2003,

    YPXL/I is a protein interaction motif recognized by <i>Aspergillus</i> PalA and its human homologue, AIP1/Alix

    , MOLECULAR AND CELLULAR BIOLOGY, Vol: 23, Pages: 1647-1655, ISSN: 0270-7306
  • Journal article
    Mangoni AA, Desai SR, Shaikh H, Barker RD, Mufti GJ, Jackson SHDet al., 2003,

    An unusual case of pneumonia

    , INTERNATIONAL JOURNAL OF CLINICAL PRACTICE, Vol: 57, Pages: 153-154, ISSN: 1368-5031
  • Journal article
    Horsefield R, Yankovskaya V, Törnroth S, Luna-Chavez C, Stambouli E, Barber J, Byrne B, Cecchini G, Iwata Set al., 2003,

    Using rational screening and electron microscopy to optimize the crystallization of succinate:ubiquinone oxidoreductase from <i>Escherichia coli</i>

    , ACTA CRYSTALLOGRAPHICA SECTION D-BIOLOGICAL CRYSTALLOGRAPHY, Vol: 59, Pages: 600-602, ISSN: 0907-4449
  • Journal article
    Kagoshima M, Ito K, Cosio B, Adcock IMet al., 2003,

    Glucocorticoid suppression of nuclear factor-κB: a role for histone modifications

    , BIOCHEMICAL SOCIETY TRANSACTIONS, Vol: 31, Pages: 60-65, ISSN: 0300-5127
    • Cite
    • Citations: 51
  • Journal article
    Yankovskaya V, Horsefield R, Törnroth S, Luna-Chavez C, Miyoshi H, Léger C, Byrne B, Cecchini G, Iwata Set al., 2003,

    Architecture of succinate dehydrogenase and reactive oxygen species generation

    , SCIENCE, Vol: 299, Pages: 700-704, ISSN: 0036-8075
  • Conference paper
    Kagoshima M, Ito K, Cosio B, Adcock IMet al., 2003,

    Glucocorticoid suppression of nuclear factor-κB: A role for histone modifications

    , Pages: 60-65, ISSN: 0300-5127

    Corticosteroids are by far the most effective treatment for chronic inflammatory diseases such as asthma. Inflammation in asthma is characterized by the increased expression of multiple inflammatory genes, including those that encode cytokines, chemokines, adhesion molecules, and inflammatory enzymes and receptors. Increased expression of inflammatory genes is regulated by pro-inflammatory transcription factors, such as nuclear factor κB (NF-κB). These bind to, and activate, co-activator molecules that then acetylate core histones resulting in elevated gene transcription. Corticosteroids reverse histone acetylation at the site of inflammatory gene transcription, either by direct binding of the activated glucocorticoid receptor to NF-κB-associated co-activators or by recruitment of histone deacetylases to the activated transcription complex. Understanding how corticosteroids work in asthma may help in designing novel corticosteroids with fewer systemic effects, as well as novel anti-inflammatory approaches.
  • Journal article
    McShane D, Davies JC, Davies MG, Bush A, Geddes DM, Alton EWFWet al., 2003,

    Airway surface pH in subjects with cystic fibrosis

    , EUROPEAN RESPIRATORY JOURNAL, Vol: 21, Pages: 37-42, ISSN: 0903-1936
    • Cite
    • Citations: 102
  • Book chapter
    Bush A, Davies JC, 2003,

    Cystic Fibrosis in Childhood

    , Respiratory Medicene 3rd Edition, Editors: Gibson, Geddes, Costabel, Sterk, Corrin, Gibson, Geddes, Costabel, Sterk, Corrin, Publisher: Saunders, Pages: 1477-1494, ISBN: 9780702026133
  • Book chapter
    Davies, Geddes, Alton, 2003,

    Pulmonary Gene Therapy

    , Pharmaceutical Gene Delivery Systems, Editors: Rolland, Sullivan, New York, Publisher: Marcel Dekker Inc., Pages: 363-396, ISBN: 9780824742355
  • Journal article
    Iwata M, Abramson J, Byrne B, Iwata Set al., 2003,

    Structure and function of quinone binding membrane proteins

    , MEMBRANE PROTEINS, Vol: 63, Pages: 151-176, ISSN: 0065-3233

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