Imperial College London

DrBalvinderHanda

Faculty of MedicineNational Heart & Lung Institute

Honorary Clinical Lecturer
 
 
 
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Contact

 

balvinder.handa05

 
 
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Location

 

Commonwealth BuildingHammersmith Campus

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Summary

 

Overview

Handa BS, Li X, Aras KK, Qureshi NA, Mann I, Chowdhury RA, Whinnett ZI, Linton NWF, Lim PB, Kanagaratnam P, Efimov IR, Peters NS, Ng FS Granger Causality-based analysis for classification of fibrillation mechanisms and localisation of rotational drivers. Circulation Arrhythmia and Electrophysiology. Jan 2020 (In Press)

Li, X., Roney, C.H., Handa, B.S. et al. Standardised Framework for Quantitative Analysis of Fibrillation Dynamics. Sci Rep 9, 16671 (2019) doi:10.1038/s41598-019-52976-y

Handa BS, Lawal S, Wright IJ, Li X, Cabello-García J, Mansfield C, et al. Interventricular Differences in Action Potential Duration Restitution Contribute to Dissimilar Ventricular Rhythms in ex vivo Perfused Hearts    [Internet]. Vol. 6, Frontiers in Cardiovascular Medicine. 2019. p. 34. Available from: https://www.frontiersin.org/article/10.3389/fcvm.2019.00034 

Handa BS, Roney CH, Houston C et al. Analytical approaches for myocardial fibrillation signals. Comput Biol Med. 2018;102:315–26.

Meijles DN, Zoumpoulidou G, Markou T, Rostron KA, Patel R, Lay K, Handa BS et al. The cardiomyocyte “redox rheostat”: Redox signalling via the AMPK-mTOR axis and regulation of gene and protein expression balancing survival and death. J Mol Cell Cardiol. 2019 Feb;

Pikkarainen S, Kennedy RA, Marshall AK, Tham el L, Lay K, Kriz TA, Handa BS, Clerk A, Sugden PH. Regulation of expression of the rat orthologue of mouse double minute 2 (MDM2) by H(2)O(2)-induced oxidative stress in neonatal rat cardiac myocytes. J Biol Chem. 2009 Oct 2;284(40):27195-210.

Conference Abstracts

Handa BS, Li X, Qureshi N, Mann I, Kanagaratnam P, Peters NS, Ng FS. Granger Causality-based analysis to accurately identify specific electrophenotypes of myocardial fibrillation (P6594). European Society of Cardiology Congress, Paris, 2019.

Handa BS, Li X, Mansfield CA, Jabbour RJ, Pitcher D, Chowdhury RA, Peters NS, Ng FS. Ventricular fibrosis spatial distribution and quantity is a key mechanistic determinant of ventricular fibrillation mechanisms (P1594). European Society of Cardiology Congress, Paris, 2019.


Handa BS, Li X, Roney C, Pitcher D, Chowdhury RA, Peters NS, Ng FS. Gap junction modulation is a major determinant of the mechanism and organisation of ventricular fibrillation and a potential therapeutic target. EP Eur [Internet]. 2019 May 3;21(Supplement_2):ii851-ii857. Available from: https://doi.org/10.1093/europace/euz084

Handa BS, Li X, Mansfield CA, Jabbour RJ, Pitcher D, Chowdhury RA, Peters NS, Ng FS. The Degree And Spatial Distribution Of Myocardial Fibrosis Influences The Underlying Mechanism Of Ventricular Fibrillation. Heart Rhythm [Internet]. 2019 May 1;16(5):S142–235. Available from: https://doi.org/10.1016/j.hrthm.2019.04.015


Handa BS, Li X, Roney C, Pitcher D, Chowdhury RA, Peters NS, Ng FS. Gap Junction Modulation Is A Major Determinant Of The Mechanism And Organisation Of Ventricular Fibrillation. Heart Rhythm [Internet]. 2019 May 1;16(5):S142–235. Available from: https://doi.org/10.1016/j.hrthm.2019.04.015


Handa BS, Li X, Mansfield CA, Jabbour RJ, Pitcher D, Chowdhury RA, Peters NS, Ng FS. BS62 Myocardial fibrosis and the degree of gap junction coupling directly modifies the underlying mechanism of fibrillation Heart 2019;105:A179-A180.


R Jabbour, K Kapnisi, D Mawad, B Handa, L Couch, C Mansfield, F Perbillini, C Terracciano, M Stevens, A Lyon, G Smith, N Peters, F S Ng, S Harding, P5705. Conductive polymers affect myocardial conduction velocity but are not pro-arrhythmic, European Heart Journal, Volume 39, Issue suppl_1, August 2018, ehy566.P5705, https://doi.org/10.1093/eurheartj/ehy566.P5705


Doshi, R., Handa, B., Balogun-Lynch, J., Sloan-Brittain, E., Gulati, M., Penn, H., & Hamdulay, S. S. (2014). Utility of fluoro-deoxy-glucose positron emission tomography/computerized tomogrophy in general rheumatology practice. Rheumatology (United Kingdom), 53, i160.