Imperial College London
Alternate

Dr Rob White

Faculty of Medicine, Department of Infectious Disease

Senior Lecturer
 
 
 
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Contact

 

+44 (0)20 7594 1124robert.e.white Website

 
 
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Location

 

308Norfolk PlaceSt Mary's Campus

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Summary

 

Publications

Citation

BibTex format

@article{Farrell:2022:10.3390/biom12010017,
author = {Farrell, PJ and White, RE},
doi = {10.3390/biom12010017},
journal = {Biomolecules},
title = {Do Epstein-Barr virus mutations and natural genome sequence variations contribute to disease?},
url = {http://dx.doi.org/10.3390/biom12010017},
volume = {12},
year = {2022}
}
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RIS format (EndNote, RefMan)

TY  - JOUR
AB  - Most of the world’s population is infected by the Epstein–Barr virus (EBV), but the incidence of the diseases associated with EBV infection differs greatly in different parts of the world. Many factors may determine those differences, but variation in the virus genome is likely to be a contributing factor for some of the diseases. Here, we describe the main forms of EBV genome sequence variation, and the mechanisms by which variations in the virus genome are likely to contribute to disease. EBV genome deletions or polymorphisms can also provide useful markers for monitoring disease. If some EBV strains prove to be more pathogenic than others, this suggests the possible value of immunising people against infection by those pathogenic strains
AU  - Farrell,PJ
AU  - White,RE
DO  - 10.3390/biom12010017
PY  - 2022///
SN  - 2218-273X
TI  - Do Epstein-Barr virus mutations and natural genome sequence variations contribute to disease?
T2  - Biomolecules
UR  - http://dx.doi.org/10.3390/biom12010017
UR  - http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000757360200001&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=1ba7043ffcc86c417c072aa74d649202
UR  - https://www.mdpi.com/2218-273X/12/1/17
UR  - http://hdl.handle.net/10044/1/97366
VL  - 12
ER  -
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