Publications
140 results found
Consonni D, De Matteis S, Pesatori AC, et al., 2012, Increased lung cancer risk among bricklayers in an Italian population-based case-control study, AMERICAN JOURNAL OF INDUSTRIAL MEDICINE, Vol: 55, Pages: 423-428, ISSN: 0271-3586
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- Citations: 8
De Matteis S, Consonni D, Pesatori AC, et al., 2012, Tobacco smoking women are not at higher risk than men for lung cancer, Publisher: AMER ASSOC CANCER RESEARCH, ISSN: 0008-5472
Rinaldi C, Salvatore E, Giordano I, et al., 2012, Predictors of Survival in a Huntington's Disease Population from Southern Italy, CANADIAN JOURNAL OF NEUROLOGICAL SCIENCES, Vol: 39, Pages: 48-51, ISSN: 0317-1671
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- Citations: 23
Cortese B, Limbruno U, De Matteis S, et al., 2011, Prolonged Bivalirudin Infusion is Associated with Enhanced ST Segment Resolution Following Primary Percutaneous Coronary Intervention, 23rd Annual Transcatheter Cardiovascular Therapeutics (TCT) Symposium, Publisher: ELSEVIER SCIENCE INC, Pages: B89-B89, ISSN: 0735-1097
Cortese B, Limbruno U, Severi S, et al., 2011, Effect of Prolonged <i>Bivalirudin</i> Infusion on ST-Segment Resolution Following Primary Percutaneous Coronary Intervention (from the PROBI VIRI 2 Study), AMERICAN JOURNAL OF CARDIOLOGY, Vol: 108, Pages: 1220-1224, ISSN: 0002-9149
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- Citations: 22
Mensi C, Sieno C, De Matteis S, et al., 2011, Incidence of malignant mesothelioma and asbestos exposure in the lombardy region, Italy, 2000-2008, Pages: 96-98, ISSN: 1592-7830
We evaluated the trend of incidence and asbestos exposure of malignant mesothelioma (MM) in 2000-2008 in the Lombardy Region (Italy). We recorded 2,816 MMs (1,793 men, 1,023 women). The age-standardized rates (x100,000/year) were 3.4 (men) and 1.4 (women) (standard population: Europe). We found a 3.0% and 0.9% increase per year of number of cases and rate, respectively. Exposure was obtained in 2,671 cases (94.9%). Occupational exposure to asbestos was found for 1,296 (72.3%) men and 377 (36.9%) women, non-occupational exposure in 141 (13.8%) women and 58 (3.2%) men. The exposure profile within gender did not vary overyears. © PI-ME. Pavia 2011.
Vercellini P, Somigliana E, Vigano P, et al., 2011, Surgical reduction of ovarian reserve in women with endometriomas, 27th Annual Meeting of the European-Society-of-Human-Reproduction-and-Embryology, Publisher: OXFORD UNIV PRESS, Pages: I41-I41, ISSN: 0268-1161
De Matteis S, Consonni D, Lubin JH, et al., 2011, Public health impact of occupational carcinogens exposure and lung cancer, Publisher: AMER ASSOC CANCER RESEARCH, ISSN: 0008-5472
Ledger WL, Vercellini P, Somigliana E, et al., 2011, INVITED SESSION, SESSION 27: AVOIDABLE LOSS OF FERTILITY, Tuesday 5 July 2011 08:30 - 09:30, Human Reproduction, Vol: 26, Pages: i41-i41, ISSN: 0268-1161
Mensi C, Sieno C, De Matteis S, et al., 2011, [Incidence of malignant mesothelioma and asbestos exposure in the Lombardy region, Italy, 2000-2008]., G Ital Med Lav Ergon, Vol: 33, Pages: 96-98, ISSN: 1592-7830
We evaluated the trend of incidence and asbestos exposure of malignant mesothelioma (MM) in 2000-2008 in the Lombardy Region (Italy). We recorded 2,816 MMs (1,793 men, 1,023 women). The age-standardized rates (x 100,000/year) were 3.4 (men) and 1.4 (women) (standard population: Europe). We found a 3.0% and 0.9% increase per year of number of cases and rate, respectively. Exposure was obtained in 2,671 cases (94.9%). Occupational exposure to asbestos was found for 1,296 (72.3%) men and 377 (36.9%) women, non-occupational exposure in 141 (13.8%) women and 58 (3.2%) men. The exposure profile within gender did not vary over years.
Danzi GB, Ceniola M, Pomidossi GA, et al., 2010, Usefulness of Primary Angioplasty in Nonagenarians With Acute Myocardial Infarction, AMERICAN JOURNAL OF CARDIOLOGY, Vol: 106, Pages: 770-773, ISSN: 0002-9149
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- Citations: 30
Vercellini P, Somigliana E, Vigano P, et al., 2010, Post-operative endometriosis recurrence: a plea for prevention based on pathogenetic, epidemiological and clinical evidence, REPRODUCTIVE BIOMEDICINE ONLINE, Vol: 21, Pages: 259-265, ISSN: 1472-6483
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- Citations: 79
Koshiol J, Rotunno M, Consonni D, et al., 2010, Lower Risk of Lung Cancer after Multiple Pneumonia Diagnoses, CANCER EPIDEMIOLOGY BIOMARKERS & PREVENTION, Vol: 19, Pages: 716-721, ISSN: 1055-9965
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- Citations: 13
Consonni D, De Matteis S, Lubin JH, et al., 2010, Lung cancer and occupation in a population-based case-control study., American Journal of Epidemiology, Vol: 171, Pages: 323-333, ISSN: 1476-6256
The authors examined the relation between occupation and lung cancer in the large, population-based Environment And Genetics in Lung cancer Etiology (EAGLE) case-control study. In 2002-2005 in the Lombardy region of northern Italy, 2,100 incident lung cancer cases and 2,120 randomly selected population controls were enrolled. Lifetime occupational histories (industry and job title) were coded by using standard international classifications and were translated into occupations known (list A) or suspected (list B) to be associated with lung cancer. Smoking-adjusted odds ratios and 95% confidence intervals were calculated with logistic regression. For men, an increased risk was found for list A (177 exposed cases and 100 controls; odds ratio = 1.74, 95% confidence interval: 1.27, 2.38) and most occupations therein. No overall excess was found for list B with the exception of filling station attendants and bus and truck drivers (men) and launderers and dry cleaners (women). The authors estimated that 4.9% (95% confidence interval: 2.0, 7.8) of lung cancers in men were attributable to occupation. Among those in other occupations, risk excesses were found for metal workers, barbers and hairdressers, and other motor vehicle drivers. These results indicate that past exposure to occupational carcinogens remains an important determinant of lung cancer occurrence.
Koshiol J, Rotunno M, Consonni D, et al., 2009, Chronic obstructive pulmonary disease and altered risk of lung cancer in a population-based case-control study., PLOS One, Vol: 4, ISSN: 1932-6203
BACKGROUND: Chronic obstructive pulmonary disease (COPD) has been consistently associated with increased risk of lung cancer. However, previous studies have had limited ability to determine whether the association is due to smoking. METHODOLOGY/PRINCIPAL FINDINGS: The Environment And Genetics in Lung cancer Etiology (EAGLE) population-based case-control study recruited 2100 cases and 2120 controls, of whom 1934 cases and 2108 controls reported about diagnosis of chronic bronchitis, emphysema, COPD (chronic bronchitis and/or emphysema), or asthma more than 1 year before enrollment. We estimated odds ratios (OR) and 95% confidence intervals (CI) using logistic regression. After adjustment for smoking, other previous lung diseases, and study design variables, lung cancer risk was elevated among individuals with a history of chronic bronchitis (OR = 2.0, 95% CI = 1.5-2.5), emphysema (OR = 1.9, 95% CI = 1.4-2.8), or COPD (OR = 2.5, 95% CI = 2.0-3.1). Among current smokers, association between chronic bronchitis and lung cancer was strongest among lighter smokers. Asthma was associated with a decreased risk of lung cancer in males (OR = 0.48, 95% CI = 0.30-0.78). CONCLUSIONS/SIGNIFICANCE: These results suggest that the associations of personal history of chronic bronchitis, emphysema, and COPD with increased risk of lung cancer are not entirely due to smoking. Inflammatory processes may both contribute to COPD and be important for lung carcinogenesis.
Vercellini P, Somigliana E, Vigano P, et al., 2009, The effect of second-line surgery on reproductive performance of women with recurrent endometriosis: A systematic review, ACTA OBSTETRICIA ET GYNECOLOGICA SCANDINAVICA, Vol: 88, Pages: 1074-1082, ISSN: 0001-6349
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- Citations: 63
Bardella MT, Elli L, De Matteis S, et al., 2009, Autoimmune disorders in patients affected by celiac sprue and inflammatory bowel disease, ANNALS OF MEDICINE, Vol: 41, Pages: 139-143, ISSN: 0785-3890
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- Citations: 24
De Matteis S, Consonni D, Bertazzi PA, 2008, Exposure to occupational carcinogens and lung cancer risk. Evolution of epidemiological estimates of attributable fraction., Acta Biomed, Vol: 79 Suppl 1, Pages: 34-42, ISSN: 0392-4203
BACKGROUND AND AIM OF THE WORK: Lung cancer is the leading cause of cancer death world-wide. Among the possible causes, occupational risk factors play a major role and are potentially preventable. We reviewed the scientific evidence about lung cancer burden due to occupation. METHODS: We reviewed the literature and selected population case-control and cohort studies which provided estimates of the proportion of lung cancers attributable to occupational carcinogens (population attributable fraction, PAF). Different methods were used to evaluate occupational exposure to suspected/known lung carcinogens: lists of high-risk occupations, job-exposure matrix (JEM), expert assessment. Only studies which adjusted for tobacco smoking were included. RESULTS: The PAFs reported by the 32 selected Italian and international studies among men vary greatly in time and space: they ranged between 0 to 40% according to different geographical prevalence of hazardous industries (e.g., basic metal industries, shipbuilding and railroad equipment manufacturing). The PAFs estimated using JEM and expert assessment were on average higher. Data for women were usually few and insufficient to calculate stable estimates. CONCLUSIONS: A significant proportion of lung cancers is attributable to occupational carcinogens. The estimates are extremely variable in time and place and mainly depend on the industrial setting of the area under study; caution is therefore required in generalizing these results to the whole country. Alternative approaches to evaluate occupational lung cancer burden among women are necessary.
Consonni D, De Matteis S, Effectiveness of COVID-19 vaccination among healthcare workers, EPIDEMIOLOGIA & PREVENZIONE, Vol: 45, Pages: 310-311, ISSN: 1120-9763
Consonni D, De Matteis S, Pesatori AC, et al., LUNG CANCER AMONG CONSTRUCTION WORKERS IN A POPULATION-BASED CASE-CONTROL STUDY, Publisher: INFERENZE SCARL, Pages: 71-71, ISSN: 1120-9763
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