Citation

BibTex format

@article{Koudstaal:2023:10.1016/j.molmed.2023.08.010,
author = {Koudstaal, T and Funke-Chambour, M and Kreuter, M and Molyneaux, PL and Wijsenbeek, MS},
doi = {10.1016/j.molmed.2023.08.010},
journal = {Trends Mol Med},
pages = {1076--1087},
title = {Pulmonary fibrosis: from pathogenesis to clinical decision-making.},
url = {http://dx.doi.org/10.1016/j.molmed.2023.08.010},
volume = {29},
year = {2023}
}

RIS format (EndNote, RefMan)

TY  - JOUR
AB - Pulmonary fibrosis (PF) encompasses a spectrum of chronic lung diseases that progressively impact the interstitium, resulting in compromised gas exchange, breathlessness, diminished quality of life (QoL), and ultimately respiratory failure and mortality. Various diseases can cause PF, with their underlying causes primarily affecting the lung interstitium, leading to their referral as interstitial lung diseases (ILDs). The current understanding is that PF arises from abnormal wound healing processes triggered by various factors specific to each disease, leading to excessive inflammation and fibrosis. While significant progress has been made in understanding the molecular mechanisms of PF, its pathogenesis remains elusive. This review provides an in-depth exploration of the latest insights into PF pathophysiology, diagnosis, treatment, and future perspectives.
AU - Koudstaal,T
AU - Funke-Chambour,M
AU - Kreuter,M
AU - Molyneaux,PL
AU - Wijsenbeek,MS
DO - 10.1016/j.molmed.2023.08.010
EP - 1087
PY - 2023///
SP - 1076
TI - Pulmonary fibrosis: from pathogenesis to clinical decision-making.
T2 - Trends Mol Med
UR - http://dx.doi.org/10.1016/j.molmed.2023.08.010
UR - https://www.ncbi.nlm.nih.gov/pubmed/37716906
VL - 29
ER -