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  • Conference paper
    Roccia E, Mikhno A, Zanderigo F, Angelini ED, Ogden RT, Mann JJ, Laine AFet al., 2015,

    Non-Invasive Quantification of Brain [<SUP>18</SUP>F]-FDG Uptake by Combining Medical Health Records and Dynamic PET Imaging Data

    , 37th Annual International Conference of the IEEE-Engineering-in-Medicine-and-Biology-Society (EMBC), Publisher: IEEE, Pages: 2243-2246, ISSN: 1557-170X
  • Conference paper
    Meiniel W, Angelini E, Olivo-Marin J-C, 2015,

    IMAGE DENOISING BY ADAPTIVE COMPRESSED SENSING RECONSTRUCTIONS AND FUSIONS

    , Conference on Wavelets and Sparsity XVI, Publisher: SPIE-INT SOC OPTICAL ENGINEERING, ISSN: 0277-786X
  • Conference paper
    Hame Y, Angelini ED, Parikh MA, Smith BM, Hoffman EA, Barr RG, Laine AFet al., 2015,

    SPARSE SAMPLING AND UNSUPERVISED LEARNING OF LUNG TEXTURE PATTERNS IN PULMONARY EMPHYSEMA: MESA COPD STUDY

    , IEEE 12th International Symposium on Biomedical Imaging, Publisher: IEEE, Pages: 109-113, ISSN: 1945-7928
  • Conference paper
    Gan Y, Angelini E, Laine A, Hendon Cet al., 2015,

    BM3D-BASED ULTRASOUND IMAGE DENOISING VIA BRUSHLET THRESHOLDING

    , IEEE 12th International Symposium on Biomedical Imaging, Publisher: IEEE, Pages: 667-670, ISSN: 1945-7928
  • Conference paper
    Hame Y, Angelini ED, Barr RG, Laine AFet al., 2015,

    EQUATING EMPHYSEMA SCORES AND SEGMENTATIONS ACROSS CT RECONSTRUCTIONS: A COMPARISON STUDY

    , IEEE 12th International Symposium on Biomedical Imaging, Publisher: IEEE, Pages: 629-632, ISSN: 1945-7928
  • Conference paper
    Meiniel W, Le Montagner Y, Angelini E, Olivo-Marin J-Cet al., 2015,

    IMAGE DENOISING BY MULTIPLE COMPRESSED SENSING RECONSTRUCTIONS

    , IEEE 12th International Symposium on Biomedical Imaging, Publisher: IEEE, Pages: 1232-1235, ISSN: 1945-7928
  • Book chapter
    Thurston TLM, holden DW, 2015,

    interactions between salmonella and the autophagy system

    , Autophagy, infection, and the immune response, Editors: jackson, swanson, Publisher: Wiley Blackwell, ISBN: 978-1-118-67764-3
  • Journal article
    Turpin J, Alais S, Marçais A, Bruneau J, Melamed A, Gadot N, Hermine O, Bangham C, Lacoste R, Mahieux Ret al., 2015,

    Traitement d’un lymphome agressif chez un babouin naturellement infecté par STLV-1

    , Revue de primatologie, Vol: 6

    <jats:p>L’infection par le virus T Lymphotrope Humain de Type 1 (HTLV-1) est associée à une lymphoprolifération maligne nommée Leucémie/Lymphome T de l’adulte (ATLL). STLV-1 est l’homologue simien de HTLV-1 et cause aussi des ATLL chez les primates non humains. Au cours du contrôle sanitaire annuel dans notre colonie de Papio anubis naturellement infectés par STLV-1 (n=45), une femelle de 9 ans fut identifiée comme présentant des troubles respiratoires, un amaigrissement prononcé, un nombre de lymphocytes supérieur à 1010/L, des métastases pulmonaires et des lésions de la peau, similaires à celles qui sont observées chez les patients souffrant d’ATLL. Ces symptômes étaient donc évocateurs d’une maladie hématologique provoquée par l’infection à STLV-1. Ce diagnostic fut confirmé par la présence d’une lymphoprolifération massive dans la biopsie d’un ganglion inguinal et par la présence de lymphocytes présentant un noyau typique en "trèfle", caractéristique des cellules ATL, dans les frottis sanguins. Comme chez les patients souffrant d’ATLL, l’animal a reçu une combinaison d’AZT (Combivir) et d’interféron alpha (viraféron, 50 μg/semaine) pendant quatre mois. La charge provirale (PVL) fut mesurée de manière hebdomadaire. En l’absence d’amélioration des symptômes cliniques, et puisque seule une très faible diminution de la charge provirale était observée, l’animal fut euthanasié. Des analyses histologiques furent réalisées et la charge provirale mesurée dans 25 organes différents. Tous les organes lymphoïdes montraient des infiltrats lymphocytaires CD3+
  • Journal article
    Jim HSL, Lin H-Y, Tyrer JP, Lawrenson K, Dennis J, Chornokur G, Chen Z, Chen AY, Permuth-Wey J, Aben KK, Anton-Culver H, Antonenkova N, Bruinsma F, Bandera EV, Bean YT, Beckmann MW, Bisogna M, Bjorge L, Bogdanova N, Brinton LA, Brooks-Wilson A, Bunker CH, Butzow R, Campbell IG, Carty K, Chang-Claude J, Cook LS, Cramer DW, Cunningham JM, Cybulski C, Dansonka-Mieszkowska A, du Bois A, Despierre E, Sieh W, Doherty JA, Dörk T, Dürst M, Easton DF, Eccles DM, Edwards RP, Ekici AB, Fasching PA, Fridley BL, Gao Y-T, Gentry-Maharaj A, Giles GG, Glasspool R, Goodman MT, Gronwald J, Harter P, Hasmad HN, Hein A, Heitz F, Hildebrandt MAT, Hillemanns P, Hogdall CK, Hogdall E, Hosono S, Iversen ES, Jakubowska A, Jensen A, Ji B-T, Karlan BY, Kellar M, Kiemeney LA, Krakstad C, Kjaer SK, Kupryjanczyk J, Vierkant RA, Lambrechts D, Lambrechts S, Le ND, Lee AW, Lele S, Leminen A, Lester J, Levine DA, Liang D, Lim BK, Lissowska J, Lu K, Lubinski J, Lundvall L, Massuger LFAG, Matsuo K, McGuire V, McLaughlin JR, McNeish I, Menon U, Milne RL, Modugno F, Thomsen L, Moysich KB, Ness RB, Nevanlinna H, Eilber U, Odunsi K, Olson SH, Orlow I, Orsulic S, Palmieri Weber R, Paul J, Pearce CL, Pejovic T, Pelttari LM, Pike MC, Poole EM, Schernhammer E, Risch HA, Rosen B, Rossing MA, Rothstein JH, Rudolph A, Runnebaum IB, Rzepecka IK, Salvesen HB, Schwaab I, Shu X-O, Shvetsov YB, Siddiqui N, Song H, Southey MC, Spiewankiewicz B, Sucheston-Campbell L, Teo S-H, Terry KL, Thompson PJ, Tangen IL, Tworoger SS, van Altena AM, Vergote I, Walsh CS, Wang-Gohrke S, Wentzensen N, Whittemore AS, Wicklund KG, Wilkens LR, Wu AH, Wu X, Woo Y-L, Yang H, Zheng W, Ziogas A, Amankwah E, Berchuck A, Georgia Chenevix-Trench on behalf of the AOCS management group 95 96, Schildkraut JM, Kelemen LE, Ramus SJ, Monteiro ANA, Goode EL, Narod SA, Gayther SA, Pharoah PDP, Sellers TA, Phelan CMet al., 2015,

    Common Genetic Variation in Circadian Rhythm Genes and Risk of Epithelial Ovarian Cancer (EOC).

    , J Genet Genome Res, Vol: 2, ISSN: 2378-3648

    Disruption in circadian gene expression, whether due to genetic variation or environmental factors (e.g., light at night, shiftwork), is associated with increased incidence of breast, prostate, gastrointestinal and hematologic cancers and gliomas. Circadian genes are highly expressed in the ovaries where they regulate ovulation; circadian disruption is associated with several ovarian cancer risk factors (e.g., endometriosis). However, no studies have examined variation in germline circadian genes as predictors of ovarian cancer risk and invasiveness. The goal of the current study was to examine single nucleotide polymorphisms (SNPs) in circadian genes BMAL1, CRY2, CSNK1E, NPAS2, PER3, REV1 and TIMELESS and downstream transcription factors KLF10 and SENP3 as predictors of risk of epithelial ovarian cancer (EOC) and histopathologic subtypes. The study included a test set of 3,761 EOC cases and 2,722 controls and a validation set of 44,308 samples including 18,174 (10,316 serous) cases and 26,134 controls from 43 studies participating in the Ovarian Cancer Association Consortium (OCAC). Analysis of genotype data from 36 genotyped SNPs and 4600 imputed SNPs indicated that the most significant association was rs117104877 in BMAL1 (OR = 0.79, 95% CI = 0.68-0.90, p = 5.59 × 10-4]. Functional analysis revealed a significant down regulation of BMAL1 expression following cMYC overexpression and increasing transformation in ovarian surface epithelial (OSE) cells as well as alternative splicing of BMAL1 exons in ovarian and granulosa cells. These results suggest that variation in circadian genes, and specifically BMAL1, may be associated with risk of ovarian cancer, likely through disruption of hormonal pathways.
  • Conference paper
    Kouranos V, Ward S, Kokosi M, Castillo D, Chua F, Maher TM, Renzoni E, Wells AUet al., 2015,

    Uncoupling Of Restrictive And Obstructive Pattern Definitions In Lung Function Tests Of Sarcoidosis Patients

    , International Conference of the American-Thoracic-Society (ATS), Publisher: AMER THORACIC SOC, ISSN: 1073-449X

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